Thesis Statement For Hiv/Aids Essay

Thesis Statement For Hiv/Aids Essay-70
Levy was one of the first researchers to isolate the AIDS virus and is Director of the Laboratory for Tumor and AIDS Virus Research at the University of California, San Francisco.This paper may be controversial, but people need to know the other side of the story The train left the station and no one is stopping to see whether we did the right thing or not.

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This person received a stem cell transplant from a donor who was genetically resistant to HIV. Levy argues that some of the directions chosen to effect a cure "are not based on well-established experimental facts," and eliminating a virus that has integrated itself into the genes of a cell requires a better understanding of the challenge. Levy says he appreciates the pilot studies involving genetic editing that could mimic the treatment used for the "Berlin patient" and believes that the most immediate chance of success is one directed at enhancing the anti-HIV response of the immune system.

“That strategy,” he explains, “could bring persistent control of HIV as seen in healthy infected long-term survivors.

We have to ask whether we are disregarding or missing what might be a much better answer because we’re grabbing at something that looks good today.

Research studies by the author cited in this article were supported by grants from the National Institutes of Health, the California State University-wide Taskforce on AIDS, the James P.

However, as the infection progresses the immune system becomes weaker, and the patient becomes more susceptible to opportunistic infections such as Kaposi’s sarcoma or tuberculosis ( is the most favourable we have come across.

However, we are unsure how much confidence to place in this figure: while it is plausible that mass media education could be an extremely cost-effective intervention, there is at least some countervailingcost-effective.) infects cells of the human immune system, destroying or impairing their function.In the early stages of infection, no symptoms are apparent.It also could lead to the development of drug-resistant viral strains.While recognizing the important success of ART, Levy calls on his colleagues to give more attention to drug toxicities and the potential emergence of drug-resistant viruses.Pendleton Charitable Trust, the Hellman Family Foundation, the Campini Fund and the Campbell Foundation.published by Elsevier’s Cell Press, is a monthly review journal that facilitates communication between groups of highly trained professionals who share the common goal of understanding and explaining the molecular basis of disease as it relates to new clinical practice.Joseph Caputo (@sciencemetro) is Media Relations Manager for Cell Press (@Cell Press News), based in Cambridge, Massachusetts.One immune cell of long-time interest to HIV/AIDS scientists is the CD8 T lymphocyte, which is primarily thought to control HIV infection by killing infected cells. Levy discusses how this cell can employ an alternative mechanism for controlling HIV infection: it can secrete factors that suppress the virus without killing the cell, and then the infected cell can continue to function but without virus production and cell death.Because this immune response handles all HIV types, it would be important in approaches aimed at enhancing immune antiviral responses and in the development of a vaccine.“It seems misguided to just look at the short-term benefit," he says. Levy argues that one of the greatest weaknesses of existing HIV-vaccine clinical trials — most reporting limited, if any, success — is that they are mainly looking to find a better antibody to neutralize the virus.On the basis of preclinical work, he believes that there are missed opportunities to invest in basic science and study other vaccine strategies, such as enhancing the antiviral responses of immune cells, particularly those of the innate immune system.

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